June 27th, 2009
by Mike T Nelson · Filed Under: Uncategorized
I just have a sec here since Jodie is stopping by this AM for a special surprise for me! I don’t know what she has planned for the day, but I am sure it will be fun.
While I was doing some early AM research (yikes, that makes me sound like an even bigger geek), I found this study below about pain and performance.
In general, pain wil limit performance. If you are in pain, the best thing you can do to boost your strength is to get out of pain! Pain becomes a huge priority in the body.
I wish the study would have actually measured strength, since the changes they describe may or may not alter strength. Since it was not measured, from this study, we don’t know.
See the following studies that did look at this though from a previous post I did
J Physiol. 2009 Jan 15;587(Pt 1):183-93. Epub 2008 Nov 17
Effects of deep and superficial experimentally induced acute pain on muscle sympathetic nerve activity in human subjects.
Prince of Wales Medical Research Institute, Sydney, NSW 2031, Australia.
studies conducted more than half a century ago have suggested that
superficial pain induces excitatory effects on the sympathetic nervous
system, resulting in increases in blood pressure (BP) and heart rate
(HR), whereas deep pain is believed to cause vasodepression. To date,
no studies have addressed whether deep or superficial pain produces
such differential effects on muscle sympathetic nerve activity (MSNA).
Using microneurography we recorded spontaneous MSNA from the common
peroneal nerve in 13 awake subjects. Continuous blood pressure was
recorded by radial arterial tonometry. Deep pain was induced by
intramuscular injection of 0.5 ml hypertonic saline (5%) into the
tibialis anterior muscle, superficial pain by subcutaneous injection of
0.2 ml hypertonic saline into the overlying skin. Muscle pain, with a
mean rating of 4.9 +/- 0.8 (S.E.M.) on a 0-10 visual analog scale (VAS)
and lasting on average 358 +/- 32 s, caused significant increases in
MSNA (43.9 +/- 10.0%), BP (5.4 +/- 1.1%) and HR (7.0 +/- 2.0%) – not
the expected decreases. Skin pain, rated at 4.9 +/- 0.6 and lasting 464
+/- 54 s, also caused significant increases in MSNA (38.2 +/- 12.8%),
BP (5.1 +/- 2.1%) and HR (5.6 +/- 2.0%). The high-frequency (HF) to
low-frequency (LF) ratio of heart rate variability (HRV) increased from
1.54 +/- 0.25 to 2.90 +/- 0.45 for muscle pain and 2.80 +/- 0.52 for
skin pain. Despite the different qualities of deep (dull and diffuse)
and superficial (burning and well-localized) pain,
CONCLUSION We conclude that
pain originating in muscle and skin does not exert a differential
effect on muscle sympathetic nerve activity, both causing an increase
in MSNA and an increase in the LF:HF ratio of HRV. Whether this holds
true for longer lasting experimental pain remains to be seen.
Any questions on this study, post them in the comments and I will get back to you there. Comments are cool.